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Invloed GSM op in slaap vallen en EEG In dit onderzoek is gekeken naar het effect op het proces van ''in slaap vallen'' van een GSM telefoon in drie mogelijke standen: praatstand, luisterstand en standby-stand. Tevens is ook een placebo gebruikt, waarbij er geen straling was. De proefpersoon weet niet in welke stand de mobiele telefoon staat. Er wordt geconcludeerd dat de proefpersonen significant later in slaap vallen zodra de telefoon in de praatstand staat, dan wanneer de telefoon in luisterstand, standby of uit-stand stond. Ook in het 1 tot 4 Hertz gebied van de EEG waren significante verschillen geconstateerd. Neurosci Lett. 2007 Jun 21;421(1):82-6. Epub 2007 May 24. Related Articles, Links Mobile phone 'talk-mode' signal delays EEG-determined sleep onset. www.ncbi.nlm.nih.gov/sites/entrez?cmd=Retrieve&db=PubMed&list_uids=17548154&dopt=Abstract Hung CS, Anderson C, Horne JA, McEvoy P. Sleep Research Centre, Loughborough University, UK. Mobile phones signals are pulse-modulated microwaves, and EEG studies suggest that the extremely low-frequency (ELF) pulse modulation has sleep effects. However, 'talk', 'listen' and 'standby' modes differ in the ELF (2, 8, and 217Hz) spectral components and specific absorption rates, but no sleep study has differentiated these modes. We used a GSM900 mobile phone controlled by a base-station simulator and a test SIM card to simulate these three specific modes, transmitted at 12.5% (23dBm) of maximum power. At weekly intervals, 10 healthy young adults, sleep restricted to 6h, were randomly and single-blind exposed to one of: talk, listen, standby and sham (nil signal) modes, for 30min, at 13:30h, whilst lying in a sound-proof, lit bedroom, with a thermally insulated silent phone beside the right ear. Bipolar EEGs were recorded continuously, and subjective ratings of sleepiness obtained every 3min (before, during and after exposure). After exposure the phone and base-station were switched off, the bedroom darkened, and a 90min sleep opportunity followed. We report on sleep onset using: (i) visually scored latency to onset of stage 2 sleep, (ii) EEG power spectral analysis. There was no condition effect for subjective sleepiness. Post-exposure, sleep latency after talk mode was markedly and significantly delayed beyond listen and sham modes. This condition effect over time was also quite evident in 1-4Hz EEG frontal power, which is a frequency range particularly sensitive to sleep onset. It is possible that 2, 8, 217Hz modulation may differentially affect sleep onset.
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